Search for other papers by Javier Pizones in
Google Scholar
PubMed
Search for other papers by Eduardo García-Rey in
Google Scholar
PubMed
compensation mechanisms try to shift our trunk posteriorly to keep our head on top of our pelvis and preserve balance. Throughout this whole process, pelvic motion (based on rotation also called version) is essential, demonstrating the crucial role of the
Search for other papers by Yingze Su in
Google Scholar
PubMed
Search for other papers by Kangming Chen in
Google Scholar
PubMed
Search for other papers by Jinyan Wu in
Google Scholar
PubMed
Search for other papers by Junfeng Zhu in
Google Scholar
PubMed
Search for other papers by Xiaodong Chen in
Google Scholar
PubMed
foot progression angle, which further suggests the existence of compensation by tibial torsion. Furthermore, decreased FV has a negative impact on the strain on the sciatic nerve during flexion ( 56 ). Overall, both increased and decreased FV can have
Search for other papers by Nicola Krähenbühl in
Google Scholar
PubMed
Search for other papers by Tamara Horn-Lang in
Google Scholar
PubMed
Search for other papers by Beat Hintermann in
Google Scholar
PubMed
Search for other papers by Markus Knupp in
Google Scholar
PubMed
the relationships between supramalleolar deformities and inframalleolar compensation mechanisms. ICMJE Conflict of interest statement None declared Funding No benefits in any form have been received or will be received from a
Hospital del Trabajador, Santiago, Chile
Search for other papers by Juan I. Cirillo Totera in
Google Scholar
PubMed
Hospital del Trabajador, Santiago, Chile
Search for other papers by José G. Fleiderman Valenzuela in
Google Scholar
PubMed
Search for other papers by Jorge A. Garrido Arancibia in
Google Scholar
PubMed
Hospital Roberto del Río, Santiago, Chile
Search for other papers by Samuel T. Pantoja Contreras in
Google Scholar
PubMed
Search for other papers by Lyonel Beaulieu Lalanne in
Google Scholar
PubMed
Search for other papers by Facundo L. Alvarez-Lemos in
Google Scholar
PubMed
those who exceed the limit, positive SVA, unbalanced. However, a more detailed evaluation may reveal that in some cases, this normality (SVA < 5 cm) has been achieved through postural compensation mechanisms, 15 determined by the particular anatomy of
Search for other papers by Hao-Ran Xu in
Google Scholar
PubMed
Search for other papers by Yong-Hui Zhang in
Google Scholar
PubMed
Search for other papers by Yi-Li Zheng in
Google Scholar
PubMed
contributors, the underlying mechanisms of how MCE improves these functions have rarely been reported. The primary aim of this study is to describe the possible improvement mechanisms of MCE on LBP from brain, biochemistry, inflammatory, and neuromuscular
Search for other papers by Koray Şahin in
Google Scholar
PubMed
Search for other papers by Alper Şükrü Kendirci in
Google Scholar
PubMed
Search for other papers by Muhammed Oğuzhan Albayrak in
Google Scholar
PubMed
Search for other papers by Gökhan Sayer in
Google Scholar
PubMed
Search for other papers by Ali Erşen in
Google Scholar
PubMed
proprioception and neuromuscular control around the joint ( Table 2 ). In cases where some of these stabilizers are defective, stability can usually be maintained by the compensation of other components. According to this concept which is called ‘functional
South West London Elective Orthopaedic Centre, UK
Search for other papers by Charles Rivière in
Google Scholar
PubMed
Search for other papers by Stefan Lazic in
Google Scholar
PubMed
Search for other papers by Louis Dagneaux in
Google Scholar
PubMed
Search for other papers by Catherine Van Der Straeten in
Google Scholar
PubMed
Search for other papers by Justin Cobb in
Google Scholar
PubMed
Search for other papers by Sarah Muirhead-Allwood in
Google Scholar
PubMed
). Abnormal SHR A harmonious (normal) SHR occurs when a healthy flexible LPC interacts with healthy flexible hips. As soon as one of these anatomical structures becomes stiff, a clinically deleterious compensatory mechanism is often initiated by the other
Search for other papers by Julien Favre in
Google Scholar
PubMed
Institute of Microengineering, Ecole Polytechnique Fédérale de Lausanne, Lausanne, Switzerland
Search for other papers by Brigitte M. Jolles in
Google Scholar
PubMed
tissue degradation. In this sense, the mechanism associating mid-stance KFM and OA seems to be related to the mechanism described for the mid-stance KAM: the KFM and KAM peaks are two parameters characterising different aspects of the loading environment
Search for other papers by Alfonso Vaquero-Picado in
Google Scholar
PubMed
Search for other papers by Raul Barco in
Google Scholar
PubMed
Search for other papers by Samuel A. Antuña in
Google Scholar
PubMed
be seen. Histopathological studies of ECRB in patients with long-standing LE have shown necrosis as well as signs of fibre regeneration. Nevertheless, additional pathophysiological mechanisms have been suggested. Painful symptomatic LE can result in
Search for other papers by Duncan Avis in
Google Scholar
PubMed
Search for other papers by Dominic Power in
Google Scholar
PubMed
mechanism of injury, time to reduction and the method of reduction, associated musculoskeletal injuries and any abnormality in the remainder of the brachial plexus ( Table 1 ). Table 1. Variables associated with high-grade axillary nerve injury