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Juan I. Cirillo Totera Clínica Universidad de los Andes, Santiago, Chile
Hospital del Trabajador, Santiago, Chile

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José G. Fleiderman Valenzuela Clínica Universidad de los Andes, Santiago, Chile
Hospital del Trabajador, Santiago, Chile

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Jorge A. Garrido Arancibia Clínica Universidad de los Andes, Santiago, Chile

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Samuel T. Pantoja Contreras Clínica Universidad de los Andes, Santiago, Chile
Hospital Roberto del Río, Santiago, Chile

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Lyonel Beaulieu Lalanne Clínica Universidad de los Andes, Santiago, Chile

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Facundo L. Alvarez-Lemos Clínica Universidad de los Andes, Santiago, Chile

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knees and subsequent dorsal flexion of the ankles ( Fig. 10 ). 26 , 30 , 31 Fig. 10 Global vision of compensatory mechanisms. The activation of these mechanisms in response to a SI (column ‘unbalanced but compensated’) is individual to

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Jie Xiang Department of Orthopedic, Taizhou Hospital of Zhejiang Province affiliated to Wenzhou Medical University, Taizhou, Zhejiang, China

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Weibo Zhao Department of Orthopedic, Taizhou Hospital of Zhejiang Province affiliated to Wenzhou Medical University, Taizhou, Zhejiang, China

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Xiao Luo Department of Radiology, Taizhou Hospital of Zhejiang Province affiliated to Wenzhou Medical University, Taizhou, Zhejiang, China

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Zhenghua Hong Department of Orthopedic, Taizhou Hospital of Zhejiang Province affiliated to Wenzhou Medical University, Taizhou, Zhejiang, China

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Hua Luo Department of Orthopedic, Taizhou Hospital of Zhejiang Province affiliated to Wenzhou Medical University, Taizhou, Zhejiang, China

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blood coagulation, followed by atrogenic procedures using a needle. The mechanisms of SSDH development are still unclear, as the subdural cavity of the spinal cord lacks a bridging vein. In patients with SSDH, the duration from the onset of hemorrhage to

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Qi-Hao Yang Department of Sport Rehabilitation, Shanghai University of Sport, Shanghai, China

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Xue-Qiang Wang Department of Sport Rehabilitation, Shanghai University of Sport, Shanghai, China
Department of Rehabilitation Medicine, Shanghai Shangti Orthopaedic Hospital, Shanghai, China

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aggravate the symptoms of the patients ( 3 ). The main inducing factors of NLBP are decreased lumbar stability and muscle strength, and poor spinal stability and posture control is an important pathological mechanism of recurrent symptoms in patients with

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